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Brain breaks: attention belongs to a living body

Attention belongs to a living body. Make room for nourishing breaks, and support the transition away from work and back again.

Watch videoMinimise video5:13Brain breaks: attention belongs to a living body

In clinical work, I see people with ADHD who spend almost the whole day trying to catch up. Some are taking medication and some are not. They move from one demand to the next, feeling that a break is the last thing they can afford. There is always something unfinished. By the time they stop, they are exhausted, frustrated and often angry with themselves. We talk about their executive functioning, but sometimes we have barely looked at whether the day contains any real recovery.

The body is an organic, biological system. It is an animal. It is not a machine. Attention belongs to that body: to living cells, chemical signalling, sleep and wakefulness, movement, nourishment, stress and recovery. If we expect the brain to work continuously and then expect medication to carry that expectation, we have the wrong view of what we are asking it to do.

I think this matters because people can interpret every difficult afternoon as evidence of a defective brain. They might have worked through lunch, barely moved, ignored thirst and spent every pause on their phone. They then try to extract another three hours of concentration from themselves. We need to understand the conditions around attention before making another judgement about the person.

Attention has a biological context

The brain is active tissue. Its cells need energy and maintain a complex system of communication. Dopamine and noradrenaline are part of the signalling involved in attention, motivation and behavioural regulation. Medication can influence those systems and help someone organise, begin and remain with an activity. That improvement takes place within the same living body, with the same need for sleep, meals, movement and periods of lower demand.

Research gives us several ways of looking at this. Adult ADHD studies have found that some aspects of attention become more inconsistent during prolonged tasks. A recent EEG study also found changing patterns of attentional preparation as a task continued. Other experiments have investigated biochemical changes during a demanding working day. These are different pieces of evidence, but together they give us reason to take the duration and conditions of effort seriously.

For the person trying to get through the day, the useful question is quite practical: what happens to your attention when you keep going? Do you begin rereading the same paragraph? Do small decisions become irritating? Does your body tighten? Are you still working, or sitting there while your mind fights the work? These observations can tell us when the current approach has stopped serving you.

Sleep, exercise and diet remain the three pillars here. A short daytime break sits within those foundations. It gives the day a different rhythm: effort, a change in demand, then a return. Research on brief breaks supports reductions in fatigue and increases in vigour. The effect on work performance varies, which is another reason to pay attention to how you actually feel and function after a break.

Take the break even when the work is unfinished

One of the most powerful things I sometimes say is: I know you have not got it done. Take a decent break anyway. Something restful. Something nourishing. When a person is already struggling, the urge is often to push harder and take away everything enjoyable until the work is finished. That can leave them with a day made almost entirely of pressure.

The difficulty is that stopping can bring up a strong feeling of guilt. You might hear, “I have already wasted enough time,” or “Other people would have finished this by now.” That inner monologue deserves attention. If it makes you ignore your body every time something runs late, it becomes part of the problem we are trying to change.

A break needs a place in the plan. If you wait until everything is complete, the break may never come. Put a pause after a manageable period of effort, or between two defined parts of a task. A timer can help you notice that the period has ended. It should also give you a moment to check what you need: more movement, quiet, food, fresh air, or a longer pause because the demand has been high.

There is an important distinction between rest and getting pulled into another activity. You may leave a difficult task and find yourself checking messages, following links and dealing with three new problems. You have stopped the original work, but the stream of demands has continued. To understand recovery, we need to examine what the break contains.

More nourishing, less detrimental

I use two simple words: nourishing and detrimental. What leaves your system more settled, more able to participate in life, or more connected? What leaves you more agitated, depleted, stuck or unable to stop? We need a crystal-clear view of our own patterns, then we need to tip the scales in our favour. More nourishing, less detrimental.

A walk around the block without your phone can be nourishing. You move your body, look beyond a screen and encounter the world at walking pace. Taking your dog out gives the pause a familiar purpose. Stretching, sitting somewhere quiet, looking at trees, or spending a little time with a friend can also change the quality of the day. A walk to a coffee shop may provide movement, contact and pleasure; notice how the coffee itself affects the rest of your day. Hiking in the forest may give you a longer period away from the demands that have been crowding your attention.

These are examples to explore. A crowded coffee shop can be exhausting for someone who needs quiet. Another person may recover through conversation after hours alone. You need to learn what nourishes you in the circumstances you are actually in. Look at the effect over time, including how readily you can return, how you feel later, and whether the activity supports the evening you want to have.

Experiments on nature and walking give this clinical idea some support. Research has examined both very brief views of greenery and longer walks, with changes in selected attention measures and perceived restoration. Workplace trials have also tested park walks and relaxation during lunch breaks. This gives us a reason to make room for ordinary contact with the world outside the task, including small opportunities that are available in a working day.

I particularly encourage people to try some breaks without the phone. A laboratory experiment found poorer subsequent task performance after a phone-based break than after comparable breaks on paper or a larger computer. In everyday life, the issue is often the endlessness of the feed: a five-minute pause becomes a new sequence of decisions, emotions and invitations to keep watching. If your usual break leaves you harder to settle and harder to return, that is useful information. Change what the pause contains and observe what follows.

Make stopping and returning easier

For ADHD, the transition itself may need support. You finally get into the work, and stopping feels risky because you do not trust yourself to return. We should take that fear seriously. Before leaving the task, write down the next physical action: “Open the document and revise the second paragraph,” or “Put the next load in the machine.” Leave the materials ready. A clear return point can reduce the amount of organising you have to do when the break ends.

Then choose a break that has a shape. Walk to the corner and back. Sit outside for a few minutes. Stretch and drink some water. Eat lunch away from the work if you can. Tell a friend you are taking a short pause and will return at an agreed time. The aim is to make a real change in demand while keeping the next step easy to recognise.

Try it with curiosity. Notice how you were feeling before the break and what changed afterwards. Perhaps you feel less tense but still need a smaller task. Perhaps the walk helped, while opening social media made returning much harder. Perhaps the timer was too frequent, or the break was too short. That information helps you adjust the arrangement. A strategy needs to fit the person, the activity and the day.

An OT or therapist can help with this process, especially when taking a break immediately becomes an argument with yourself. We can look at the practical sequence and the psychological pattern together: what makes stopping difficult, what pulls the pause off course, and what helps you return without aggression towards yourself. Support may also mean negotiating an actual lunch break or a more workable pattern of demands. The environment needs to allow the change you are trying to make.

If medication has improved your focus, use some of that improved organisation to look after the body. Remember the walk. Make room for movement. Finish the working day with a clear next step written down, then leave time to eat and prepare for sleep. Enhanced focus can help you participate in nourishing activities too. It should have a place in the whole life you are trying to build.

This is how I use the SEEDS model. Attention, the body, the activity and the surrounding conditions belong in the same picture. We can take ownership by changing those conditions a little at a time. Trust the nourishing things: give them a repeated place in the day, observe their effects and keep adjusting. A biological system needs care during the day it is being asked to work. Give your brain breaks.

The evidence behind the article

Research notes and references

How to read this article. The first-person observations and the nourishing/detrimental formulation are the author's clinical account. Research supports important components; no trial has tested this entire SEEDS brain-break approach. The strongest direct adult ADHD evidence here concerns sustained attention, neurochemistry, sleep and exercise. Much of the experimental break and nature evidence comes from adults without an ADHD diagnosis. The practical arrangements for stopping and returning are clinical applications.

  1. Changing attention over time. Reference 2 studied 29 diagnosed adults not taking stimulants and 30 controls, using four 20-minute tasks. Greater decline appeared in selected outcomes, with no flexibility difference. Reference 3 studied EEG during a slow-paced task in 24 adults with ADHD and 20 controls; it did not test a break intervention. Their findings support assessing sustained demands, without prescribing a universal work interval.

  2. Biology and medication. Reference 11 linked dopamine responses to symptom improvement in a small prospective ADHD sample. Reference 12 examined dopamine and noradrenaline transporter binding before and during methylphenidate treatment. Neither tested rest breaks. The recommendation to retain recovery in a medicated day is a clinical application of whole-person care; it is not advice to interrupt medication or alter its timing.

  3. Fatigue mechanisms. Reference 10 compared high- and low-demand cognitive work over roughly six hours. It provides preliminary metabolic evidence, not proof that an ordinary break replenishes dopamine, removes toxic metabolites or reverses cellular damage. Reference 4 offers a different explanation: brief goal changes prevented a vigilance decline, challenging a simple exhausted-attention-tank model. “Recharge” or “reset” should therefore remain everyday descriptions of a felt recovery or change in engagement, not a measured neurotransmitter claim.

  4. What brief breaks improve. Reference 1 pooled experimental and quasi-experimental studies and found small benefits for fatigue and vigour, without a significant overall performance benefit. Several component studies, including reference 9, are already represented in that review and must not be counted as independent additional replications. Reference 5 was observational, with preferred activities and earlier breaks associated with recovery.

  5. Nature and ordinary recovery. Reference 7 was a randomised trial in healthy adults: the nature-specific result was an EEG measure and greater perceived restoration; behavioural executive control improved in both walking groups. Reference 8 tested lunch breaks in workers, with modest and season-dependent effects. Reference 9 concerned briefly viewing a roof image, not being outdoors. The dog, friend, stretching and coffee-shop examples are suggestions to evaluate, rather than activities shown to restore every adult with ADHD.

  6. Phone-free breaks. Reference 6 randomised students to a hypothetical shopping break on different media or no break. It did not test social-media feeds, dopamine pathways or adults selected for ADHD. Using it to support a trial of phone-free recovery is a clinical inference. The article's test is the person's actual pattern after the pause.

  7. Existing evidence in the main collection. Sleep-loss experiments and observed sleepiness are covered by reference 13 and reference 14; fatigue outcomes from added sleep treatment by reference 15; acute exercise by reference 16; supported longer-term exercise by reference 17. These support the broader biological context. They do not show that missing a single meal or short break causes ADHD, or that a break substitutes for adequate sleep.

References: 1, 2, 3, 4, 5, 6, 7, 8, 9, 10, 11, 12, 13, 14, 15, 16, 17, 18.

  1. Albulescu, P., Macsinga, I., Rusu, A., Sulea, C., Bodnaru, A., & Tulbure, B. T. (2022). ‘Give me a break!’ A systematic review and meta-analysis on the efficacy of micro-breaks for increasing well-being and performance. PLOS ONE, 17(8), e0272460. doi:10.1371/journal.pone.0272460.

    Read source 1
    Study notes

    Systematic review of 22 experimental/quasi-experimental samples (2, 335 participants), healthy workers/students rather than diagnosed ADHD. Breaks of up to ten minutes gave small benefits for vigour (d=.36) and fatigue (d=.35); overall performance benefit was not significant (d=.16, p=.116). Performance depended on task and duration; no standard optimal interval. Effects mainly immediate, with limited follow-up and variable bias. Full publisher methods/results inspected. This is synthesis, not a new clinical trial; reference 9 is one of its included experiments, so it is not an independent replication.

  2. Tucha, L., Fuermaier, A. B. M., Koerts, J., et al. (2017). Sustained attention in adult ADHD: time-on-task effects of various measures of attention. Journal of Neural Transmission, 124(Suppl 1), 39–53. doi:10.1007/s00702-015-1426-0. First published online 24 July 2015.

    Read source 2
    Study notes

    Clinical comparison: 29 diagnosed adults with ADHD, none currently taking stimulants (two taking antidepressants), and 30 controls. Four 20-minute tests with counterbalanced order and breaks between tests. Greater time-on-task deterioration in selected alertness, selective/divided attention outcomes; flexibility differences absent. Some attention outcomes were null. Small sample, comorbidity and multiple comparisons limit interpretation. Full publisher methods/results inspected. Break effectiveness was not manipulated; this is evidence to assess sustained demands, not proof that a break repairs ADHD or that all attention declines uniformly.

  3. Rasoulzadeh, V., Buyck, I., & Wiersema, R. (2026). Altered pre-stimulus alpha dynamics index sustained attention difficulties in ADHD. International Journal of Psychophysiology, 226, 113401. doi:10.1016/j.ijpsycho.2026.113401. First published online 3 May 2026.

    Read source 3
    Study notes

    EEG/behavioural study during a slow-paced two-choice task: 24 adults with ADHD (mean age 34.38) and 20 controls (mean 36.55). ADHD showed a steeper time-on-task rise in posterior pre-stimulus alpha and less sustained anticipatory alpha suppression. Lower alpha predicted faster responses in controls, but not ADHD. Primary publisher abstract/introduction and PubMed metadata inspected; full methods unavailable, so medication status and unreported allocation details are not inferred. Alpha is a physiological proxy, not a neurotransmitter measurement, biological energy gauge or demonstrated response to a rest intervention.

  4. Ariga, A., & Lleras, A. (2011). Brief and rare mental ‘breaks’ keep you focused: deactivation and reactivation of task goals preempt vigilance decrements. Cognition, 118(3), 439–443. doi:10.1016/j.cognition.2010.12.007.

    Read source 4
    Study notes

    84 students randomised to four conditions during a 40-minute visual-vigilance task. Two brief memory-goal switches at 20/30 minutes prevented decline in target-detection sensitivity; the response-time interaction was null. The ‘break’ was an active digit-memory task, not relaxation or exercise. Scientific article-in-press PDF fully inspected on an external host; publication metadata verified in PubMed. Supports controlled disengagement/re-engagement and goal-reactivation theory, challenging a simple resource-depletion account. No ADHD selection, durable flexibility training, Pomodoro schedule comparison or chemical reset was tested.

  5. Hunter, E. M., & Wu, C. (2016). Give me a better break: Choosing workday break activities to maximize resource recovery. Journal of Applied Psychology, 101(2), 302–311. doi:10.1037/apl0000045. First published online 10 August 2015.

    Read source 5
    Study notes

    Experience-sampling study of 95 employees over five working days. Preferred activities and earlier breaks were associated with greater self-reported resource recovery; longer pauses or frequent short pauses were associated with more recovery than infrequent short ones. Observational: preference/timing may reflect circumstances, and causal superiority of one break is not established. Primary abstract verified; full publisher methods unavailable. Supports attending to choice, timing and frequency, rather than forcing an activity on everyone. Not an ADHD sample, neurochemical experiment or universal morning-break prescription.

  6. Kang, S., & Kurtzberg, T. R. (2019). Reach for your cell phone at your own risk: The cognitive costs of media choice for breaks. Journal of Behavioral Addictions, 8(3), 395–403. doi:10.1556/2006.8.2019.21.

    Read source 6
    Study notes

    414 students randomised to a hypothetical-shopping break on a phone, computer or paper, or no break, between anagram tasks. Phone-break participants subsequently performed less efficiently and solved fewer anagrams than other break-media groups. ‘Cognitive depletion’ was inferred from task performance, not measured cellular exhaustion. Primary indexed full methods/discussion inspected; direct PMC retrieval challenged by browser access check. This specific laboratory activity does not establish that all screen uses are harmful, that scrolling causes a measured dopamine change, or that the finding generalises to diagnosed ADHD.

  7. McDonnell, A. S., & Strayer, D. L. (2024). Immersion in nature enhances neural indices of executive attention. Scientific Reports, 14, 1845. doi:10.1038/s41598-024-52205-1.

    Read source 7
    Study notes

    92 healthy adults (18–57) randomised to a 40-minute nature or urban walk, with attention-task/EEG testing before/after. Nature was perceived as more restorative and produced a larger error-related negativity response, an EEG proxy of performance monitoring. Behavioural executive-control indices improved in both groups; there was no nature-specific behavioural advantage. Primary indexed publisher methods/discussion inspected; direct PDF opening blocked by publisher authentication redirect. Supports selected restoration outcomes after a substantial walk, not a proven ADHD treatment or dopamine reset. A second McDonnell 2024 resting-EEG report may overlap this trial and should not automatically be counted as independent replication.

  8. De Bloom, J., Sianoja, M., Korpela, K., Tuomisto, M., Lilja, A., Geurts, S., & Kinnunen, U. (2017). Effects of park walks and relaxation exercises during lunch breaks on recovery from job stress: Two randomized controlled trials. Journal of Environmental Psychology, 51, 14–30. doi:10.1016/j.jenvp.2017.03.006.

    Read source 8
    Study notes

    153 Finnish knowledge workers randomised to 15-minute lunchtime park walks, relaxation or usual breaks, for ten workdays. Spring/fall cohorts differed: little recovery benefit in spring; fall restoration/fatigue effects were modest and short-lived (reported within-person d=.22–.58). Both interventions reduced tension. Main source compares groups over time; the quoted d values are not all between-group effects. Primary publisher methods/results and university author copy inspected. No ADHD selection or neurotransmitter assay. Related papers from the same trial should not be counted as independent evidence.

  9. Lee, K. E., Williams, K. J. H., Sargent, L. D., Williams, N. S. G., & Johnson, K. A. (2015). 40-second green roof views sustain attention: The role of micro-breaks in attention restoration. Journal of Environmental Psychology, 42, 182–189. doi:10.1016/j.jenvp.2015.04.003.

    Read source 9
    Study notes

    150 university students randomised to a 40-second image of a flowering green roof or concrete roof between attention tasks. Green viewing was associated with selected lower response variability and fewer omissions later in the subsequent task; the omission difference was small (r=.14) and absent in its first half. Scientific publisher PDF inspected on an external host. No no-break group, diagnosed ADHD sample, field walk or neurochemical measurement. Included in reference 1's synthesis, so not independent additional pooled evidence. Supports brief visual changes as an option to explore, not a universal restoration dose.

  10. Wiehler, A., Branzoli, F., Adanyeguh, I., Mochel, F., & Pessiglione, M. (2022). A neuro-metabolic account of why daylong cognitive work alters the control of economic decisions. Current Biology, 32(16), 3564–3575.e5. doi:10.1016/j.cub.2022.07.010.

    Read source 10
    Study notes

    40 healthy adults (24 high-demand; 16 low-demand) performed cognitive work over 6.25 hours, with two 10-minute pauses. MRS measured glutamate concentration and glutamate/glutamine diffusion in lateral prefrontal/visual cortex. The concentration interaction was driven mainly by a decrease in the easy group; diffusion increased in the hard-group prefrontal region. Choices shifted toward lower-cost options. The extracellular-accumulation/cognitive-cost mechanism is inferred; causal toxicity or tissue damage was not shown. Rest/sleep recovery remained an explicit research question. Full primary scientific PDF, including STAR methods, inspected on an external host after publisher access failed. No ADHD sample, randomised break comparison or neurotransmitter reset after walking.

  11. Volkow, N. D., Wang, G.-J., Tomasi, D., et al. (2012). Methylphenidate-elicited dopamine increases in ventral striatum are associated with long-term symptom improvement in adults with attention deficit hyperactivity disorder. Journal of Neuroscience, 32(3), 841–849. doi:10.1523/JNEUROSCI.4461-11.2012.

    Read source 11
    Study notes

    Prospective PET study of 20 treatment-naïve adults with ADHD before and after 12 months of individually titrated oral methylphenidate. Dopamine responses to an intravenous challenge, inferred through raclopride binding, were associated with reduced inattention after clinical treatment. Primary abstract verified. Small, uncontrolled sample and different challenge/clinical routes constrain interpretation. Shows a relationship between drug-associated dopamine signalling and symptom response; it neither measures a break's effect nor establishes that ordinary fatigue is a depleted dopamine supply. No dose or medication-timing advice is derived.

  12. Oya, M., Matsuoka, K., Kubota, M., et al. (2026). Effects of extended-release methylphenidate on dopamine and norepinephrine transporters in adults with attention-deficit/hyperactivity disorder: A longitudinal dual-tracer PET study. Psychiatry and Clinical Neurosciences, 80(1), 48–54. doi:10.1111/pcn.13911. First published online 23 October 2025.

    Read source 12
    Study notes

    Longitudinal PET/cognitive study: 21 diagnosed adults at baseline and 12 reassessed after treatment stabilisation. Methylphenidate reduced dopamine/noradrenaline transporter binding in selected regions; 3 of 10 cognitive measures improved. Binding-change magnitude did not correlate with cognitive change. Indexed primary full methods/results and publisher metadata inspected. Small completer sample, uncontrolled repeated testing, attrition and multiple measures limit causal interpretation. Transporter binding is not a direct synaptic-concentration measurement. Supports medication's biological action in both systems, while leaving the need for rest to clinical whole-person reasoning. No break, fatigue-recovery or sleep-aid intervention was tested.

  13. Dan O, Cohen A, Asraf K, Saveliev I, Haimov I. The Impact of Sleep Deprivation on Continuous Performance Task Among Young Men With ADHD. Journal of Attention Disorders. 2021;25(9):1284–1294. doi:10.1177/1087054719897811. First published online January 9, 2020.

    Read source 13
    Study notes

    34 young men: 16 with ADHD and 18 without ADHD; mean age 25.38 years. Sleep deprivation worsened missed responses, incorrect responses, reaction time and response variability in both groups. The ADHD group performed worse overall, but the published study concluded that both groups were similarly affected by deprivation. Small male sample, extreme acute sleep loss and repeated task testing. This is direct adult ADHD evidence for impaired attention after sleep deprivation; it does not quantify the impact of ordinary short nights, establish worsening across every clinical ADHD symptom or show greater vulnerability in all ADHD adults.

  14. Helfer B, Bozhilova N, Cooper RE, Douzenis JI, Maltezos S, Asherson P. The key role of daytime sleepiness in cognitive functioning of adults with attention deficit hyperactivity disorder. European Psychiatry. 2020;63(1):e31. doi:10.1192/j.eurpsy.2020.28.

    Read source 14
    Study notes

    111 adults, including 81 with diagnosed ADHD and 30 controls; mean overall age 32.4 years. ADHD participants withheld stimulant medication for at least 48 hours before testing. Adults with ADHD were sleepier during the attention task. Within ADHD participants, observable sleepiness correlated with missed responses even after accounting for ADHD symptom severity. Sleepiness deserves assessment when someone appears inattentive or inconsistent. Sleep was not experimentally improved or restricted, so this does not establish that sleep loss caused ADHD or that treating sleep resolves ADHD. Observer-rated sleepiness is distinct from the cause of that sleepiness; resting EEG slowing was not explained simply by normal drowsiness.

  15. Van der Ham, M., Bijlenga, D., Molenaar, N., et al. (2026). The Effects of Sleep Treatment on Symptoms of ADHD, Sleep Quality, Fatigue, and Depressive Symptoms in Adults. Journal of Attention Disorders, 30(3), 354–369. doi:10.1177/10870547251379103. First published online 27 October 2025.

    Read source 15
    Study notes

    Preliminary open-label randomized trial: 70 adults with diagnosed ADHD and a positive sleep-disorder screen. Added sleep treatment improved subjective sleep quality (between-group β=-1.98; d=0.42) and fatigue (β=-6.52; d=1.59) beyond usual ADHD care, without significant additional ADHD-symptom reduction. Twenty percent missed final assessment. Abstract and appendix read; appendix gives 25/22/23 participants, correcting the abstract's inconsistent 25/22/33. Supports treating sleep as a meaningful clinical outcome in its own right.

  16. Kuo, H.-I., Nitsche, M. A., Wu, Y.-T., Chang, J.-C., & Yang, L.-K. (2024). Acute aerobic exercise modulates cognition and cortical excitability in adults with attention-deficit hyperactivity disorder (ADHD) and healthy controls. Psychiatry Research, 340, 116108. doi:10.1016/j.psychres.2024.116108.

    Read source 16
    Study notes

    Primary PubMed/publisher abstract inspected; full methods unavailable, so allocation details and age range are not inferred. 26 drug-naïve adults with ADHD and 26 matched controls underwent assessment around 30 minutes of aerobic exercise or a control intervention. In ADHD, selected inhibitory-control/motor-learning outcomes and short intracortical inhibition improved; their changes correlated. Cortical-excitability proxies do not establish a directly measured GABA mechanism, durable symptom benefit or effects in ordinary daily tasks.

  17. Svedell, L. A., Lindvall, M. A., Holmqvist, K. L., Cao, Y., & Msghina, M. (2025). Physical exercise as add-on treatment in adults with ADHD – the START study: a randomized controlled trial. Frontiers in Psychiatry, 16, 1690216. doi: 10.3389/fpsyt.2025.1690216.

    Read source 17
    Study notes

    Adult twelve-week trial favoured supported mixed exercise added to usual care. High attrition, a small single-centre sample, unblinded participation and extra planning support for some participants limit confidence. Full intervention methods, results and limitations inspected.

  18. Solanto MV, Marks DJ, Wasserstein J, Mitchell K, Abikoff H, Alvir JMJ, Kofman MD. Efficacy of Meta-Cognitive Therapy for Adult ADHD. American Journal of Psychiatry. 2010;167(8):958–968. doi:10.1176/appi.ajp.2009.09081123.

    Read source 18
    Study notes

    88 clinically referred adults with diagnosed ADHD, stratified by medication use. Skills-focused therapy improved inattention more than supportive psychotherapy. Sessions practised task breakdown and planning, then reviewed practical and emotional obstacles to implementation. Self-esteem, depression and anxiety did not improve more than in the comparison group. Some organisation measures showed only trends. This tested a treatment package, not individual strategies.